Lady Gaga Launches "Born This Way Foundation" To Promote Safety, Skills, and Opportunity for Young People

Today, in collaboration with Harvard University and other partners, Lady Gaga is officially launching her "Born This Way Foundation".  As discussed on this blog, on news outlets, and in the peer reviewed literature, bullying is a serious problem with potential connection to public health issues such as depression and suicide.  Lady Gaga has been a vocal anti-bullying advocate, and in 2011 channeled that energy into the development of a Foundation.  Lady Gaga and her mother Cynthia Germanotta, founded the Born This Way Foundation to foster a more accepting society, where differences are embraced and individuality is celebrated.

The launch is being held at Harvard University’s Sanders Theatre.  Lady Gaga and her mother will be joined on stage by Oprah Winfrey, author and speaker Deepak Chopra, U.S. Secretary of Health and Human Services Kathleen Sebelius, and esteemed Harvard Law School professor Charles Ogletree to discuss the vision of the organization, and how we can all get involved.

An academic symposium has been planned in conjunction with the launch.  The event invites researchers, policymakers, and foundation representatives to discuss ways to reach youth and promote a culture free from bullying.  I'm proud to see that my former employer, the Education Development Center, has been invited to the symposium and will share insights from their extensive research in this area.

I look forward to hearing more about the vision of the organization, because although Lady Gaga is hugely influential (at the time of this post, she had 19,773,280 followers on twitter), the strategies and tangible goals for the Foundation are not completely clear from their website.  Under their mission statement, they list three pillars of the Foundation:

1. Safety
2. Skills
3. Opportunity

There is some discussion under those pillars of offering support to individuals through online communities, providing training in advocacy and community engagement, and providing opportunities to bring the national BTWF activities to the local level.  However, there are no details regarding how these strategies will actually be executed.  If you click on "Do Your Part" and "Review The Plan", it simply takes you back to the mission statement.

What strategies do you think could help BTWF support the three pillars of safety, skills, and opportunity?
What outcomes should they look at in order to evaluate the effectiveness of their efforts?

Gaining muscle and losing fat at the same time: If I can do it, anyone can

The idea of gaining muscle and losing fat at the same time seems impossible because of three widely held misconceptions: (a) to gain muscle you need a calorie surplus; (b) to lose fat you need a calorie deficit; and (c) you cannot achieve a calorie surplus and deficit at the same time.

Not too long ago, unfortunately I was in the right position to do some self-experiments in order to try to gain muscle and concurrently lose fat, without steroids, keeping my weight essentially constant (within a range of a few lbs). This was because I was obese, and then reached a point in the fat loss stage where I could stop losing weight while attempting to lose fat. This is indeed difficult and slow, as muscle gain itself is slow, and it apparently becomes slower as one tries to restrict fat gain. Compounding that is the fact that self-experimentation invariably leads to some mistakes.

The photos below show how I looked toward the end of my transformation from obese to relatively lean (right), and then about 1.5 years after that (left). During this time I gained muscle and lost fat, in equal amounts. How do I know that? It is because my weight is the same in both photos, even though on the left my body fat percentage is approximately 5 points lower. I estimate it to be slightly over 12 percent (on the left). This translates into a difference of about 7.5 lbs, of “fat turning into muscle”, so to speak.

A previous post on my transformation from obese to relatively lean has more measurement details (). Interestingly, I am very close to being overweight, technically speaking, in both photos above! That is, in both photos I have a body mass index that is close to 25. In fact, after putting on even a small amount of muscle, like I did, it is very easy for someone to reach a body mass index of 25. See the table below, from the body mass index article on Wikipedia ().

As someone gains more muscle and remains lean, approaching his or her maximum natural muscular potential, that person will approach the limit between the overweight and obese areas on the figure above. This will happen even though the person may be fairly lean, say with a body fat percentage in the single digits for men and around 14-18 percent for women. This applies primarily to the 5’7’’ – 5’11’’ range; things get somewhat distorted toward the extremes.

Contrast this with true obesity, as in the photo below. This photo was taken when I was obese, at the beach. If I recall it properly, it was taken on the Atlantic City seashore, or a beach nearby. I was holding a bottle of regular soda, which is emblematic of the situation in which many people find themselves in today’s urban societies. It reminds me of a passage in Gary Taubes’s book “Good Calories, Bad Calories” (), where someone who had recently discovered the deliciousness of water sweetened with sugar wondered why anyone “of means” would drink plain water ever again.

Now, you may rightfully say that a body composition change of about 7.5 lbs in 1.5 years is pitiful. Indeed, there are some people, typically young men, who will achieve this in a few months without steroids. But they are relatively rare; Scooby has a good summary of muscle gain expectations (). As for me, I am almost 50 years old, an age where muscle gain is not supposed to happen at all. I tend to gain fat very easily, but not muscle. And I was obese not too long ago. My results should be at the very low end of the scale of accomplishment for most people doing the right things.

By the way, the idea that muscle gain cannot happen after 40 years of age or so is another misconception; even though aging seems to promote muscle loss and fat gain, in part due to natural hormonal changes. There is evidence that many men may experience of low point (i.e., a trough) in their growth hormone and testosterone levels in their mid-40s, possibly due to a combination of modern diet and lifestyle factors. Still, many men in their 50s and 60s have higher levels ().

And what are the right things to do if one wants to gain muscle and lose fat at the same time? In my next post I will discuss the misconceptions mentioned at the beginning of this post, and a simple approach for concurrently gaining muscle and losing fat. The discussion will be based on my own experience and that of several HCE () users. The approach relies heavily on individual customization; so it will probably be easier to understand than to implement. Strength training is part of this simple strategy.

One puzzling aspect of strength training, from an evolutionary perspective, is that people tend to be able to do a lot more of it than is optimal for them. And, when they do even a bit more than they should, muscle gain stalls or even regresses. The minimalists frequently have the best results.

Rainn Wilson Apologizes for "Poorly Conceived Date-Rape Joke": What Would RAINN Think?

When I saw a twitter headline today that read, "Rainn Wilson Apologizes for "Poorly Conceived Date-Rape Joke", I first thought, "Is there ever a well conceived date-rape joke?"  Next I thought, "Well- that's certainly a coincidence."

The coincidence being that the other RAINN (Rape, Abuse, and Incest National Network) plays an important roll in calling out celebrities (and educating them) regarding the consequences of throwing around the term "rape".  The misuse of the term seems to be hitting epidemic proportions lately:

•   In June 2010, "Twilight" actress Kristen Stewart compared the constant scrutiny that she receives from the paparazzi to a woman being raped.  "What you don't see are the cameras shoved in my face and the bizarre intrusive questions being asked, or the people falling over themselves, screaming and taunting to get a reaction," she said of the constant paparazzi crush. "The photos are so ... I feel like I'm looking at someone being raped. A lot of the time I can't handle it. It's f---ed. I never expected that this would be my life." Stewart later apologized.  

•  In October 2011, actor Johnny Depp compared having his picture taken to being raped.  “You just feel like you’re being raped somehow. Raped. The whole thing,” he told Vanity Fair. “It feels like a kind of weird—just weird, man… Whenever you have a photo shoot or something like that, it’s like – you just feel dumb. It’s just so stupid.”  Depp later apologized.

• In November 2011, "Jersey Shore" cast mate Vinny released a song about raping strippers.  I will not repeat the lyrics here.  Vinny later apologized.

• In January 2012, actress Kim Novak took out an ad in Variety magazine to declare that she felt raped by the makers of the Oscar nominated film "The Artist" because the film used a score from one of her old movies.  The difference here:  Novak's team is not apologizing. 

Whether we like it or not, celebrities influence social norms.  When celebrities use the term "rape" as a joke or to describe their dislike of having their picture taken or their work borrowed, it cheapens what it really means.  It is meant to describe a violent act against women, men, and children...and it is a serious public health issue.  As with any public health issue, it is influenced at multiple levels (individual, interpersonal, community, society). The language we use everyday (whether to friends, family, co-workers, twitter followers, facebook friends) impacts our society on every level.  So think about the words that you choose and do your part to change the conversation.  

National Sexual Assault Hotline:  1-800-656-HOPE (4673)

The “pork paradox”? National pork consumption and obesity

In my previous post () I discussed some country data linking pork consumption and health, analyzed with WarpPLS (). One of the datasets used, the most complete, contained data from Nationmaster.com () for the following countries: Australia, Brazil, Canada, China, Denmark, France, Germany, Hong Kong, Hungary, Japan, Mexico, Poland, Russia, Singapore, Spain, Sweden, United Kingdom, and United States. That previous post also addressed a study by Bridges (), based on country-level data, suggesting that pork consumption may cause liver disease.

In this post we continue that analysis, but with a much more complex model containing the following country variables: wealth (PPP-adjusted GNP/person), pork consumption (lbs/person/year), alcohol consumption (liters/person/year), obesity (% of population), and life expectancy (years). The model and results, generated by WarpPLS, are shown on the figure below. (See notes at the end of this post.) These results are only for direct effects.

WarpPLS also calculates total effects, which are the effects of each variable on any other variable to which it is linked directly and/or indirectly. Two variables may be linked indirectly, through various paths, even if they are not linked directly (i.e., have an arrow directly connecting them). Another set of outputs generated by the software are effect sizes, which are calculated as Cohen’s f-squared coefficients. The figure below shows the total effects table. The values underlined in red are for total effects that are both statistically significant and also above the effect size threshold recommended by Cohen to be considered relevant (f-squared > 0.02).

As I predicted in my previous post, wealth is positively associated with pork consumption. So is alcohol consumption, and more strongly than wealth; which is consistent with a study by Jeanneret and colleagues showing a strong association between alcohol consumption and protein rich diets (). The inclusion of wealth in the model, compared with the model without wealth in the previous post, renders the direct and total effects of alcohol and pork consumption on life expectancy statistically indistinguishable from zero. (This often happens when a confounder is added to a model.)

Pork consumption is negatively associated with obesity, which is interesting. So is alcohol consumption, but much less strongly than pork consumption. This does not mean that if you eat 20 doughnuts every day, together with 1 lb of pork, you are not going to become obese. What this does suggest is that maybe countries where pork is consumed more heavily are somewhat more resistant to obesity. Here it should be noted that pork is very popular in Asian countries, which are becoming increasingly wealthy, but without the widespread obesity that we see in the USA.

But it is not the inclusion of Asian countries in the dataset that paints such a positive picture for pork consumption vis-à-vis obesity, and even weakens the association between wealth and obesity so much as to make it statistically non-significant. Denmark is a wealthy country that has very low levels of obesity. And it happens to have the highest level of pork consumption in the whole dataset: 142.6 lbs/person/year. So we are not talking about an “Asian paradox” here.

More like a “pork paradox”.

Finally, as far as life expectancy is concerned, the key factors seem to be wealth and obesity. Wealth has a major positive effect on life expectancy, while obesity has a much weaker negative effect. Well, access to sanitation, medical services, and other amenities of civilization, still trumps obesity in terms of prolonging life; however miserable life may turn out to be. The competing effects of these two variables (i.e., wealth and obesity) were taken into consideration, or controlled for, in the calculation of total effects and effect sizes.

The fact that pork consumption is negatively associated with obesity goes somewhat against the idea that pork is inherently unhealthy; even though pork certainly can cause disease if not properly prepared and/or cooked, which is true for many other plant and animal foods. The possible connection with liver problems, alluded to in the previous post, is particularly suspicious in light of these results. Liver diseases often impair that organ’s ability to make glycogen based on carbohydrates and protein; that is, liver diseases frequently lead to liver insulin resistance. And obesity frequently follows from liver insulin resistance.

Given that pork consumption appears to be negatively associated with obesity, it would be surprising if it was causing widespread liver disease, unless its relationship with liver disease was found to be nonlinear. (Alcohol consumption seems to be nonlinearly associated with liver disease.) Still, most studies that suggest the existence of a causal link between pork consumption and liver disease, like Bridges’s (), hint at a linear and dose-dependent relationship.

Notes

- Country-level data is inherently problematic, particularly when simple models are used (e.g., a model with only two variables). There are just too many possible confounders that may lead to the appearance of causal associations.

- More complex models ameliorate the above situation somewhat, but bump into another problem associated with country-level data – small sample sizes. We used data from 18 countries in this analysis, which is more than in the Bridges study. Still, the effective sample size here (N=18) is awfully small.

- There were some missing values in this dataset, which were handled by WarpPLS employing the most widely used approach in these cases – i.e., by replacing the missing values with the mean of each column. The percentages of missing values per variable (i.e., column) were: alcohol consumption: 27.78%; life expectancy: 5.56%; and obesity: 33.33%.

Chris Brown's Return to the Grammys: The Other Public Health Story

Sunday's Grammys were a mixture of high and low notes (pun intended), as the joy was often overshadowed by the death of Whitney Houston.  As someone who grew up in the 80's singing her songs, I too was quite shocked and saddened.  Despite broad speculation of an overdose, I'll wait to post on Whitney until we have a confirmed cause of death.

However, there was a second public health story on Sunday night.  It was the three year anniversary of Chris Brown assaulting Rihanna...and he made his return to the stage.  While Grammy executive producer Ken Ehrlich has defended the decision to bring Brown back and believes that he deserves a second chance, the warm sentiment was certainly not felt by all.  I personally turned the TV off when he began his performance.  My facebook and twitter feed blew up with comments from friends and colleagues like, "I'm no math wiz, but I'm pretty sure domestic abuser + 2 years= still a domestic abuser".  Many celebrities (both Grammy attendees and beyond) voiced the same sentiments on twitter.  For example, Wil Wheaton said simply, "everyone who apparently forgot what Chris Brown did to Rihanna should read the police report on exactly what he did."

Do you think that Chris Brown was worthy of a second chance at the Grammys? 

In addition to a discussion of giving second chances, remembering, or forgetting what he did...we must also discuss those who actually encourage what he did.  While there were outraged tweets during his performance, there were also those providing support.  The tweets that I found most disturbing were those from young women poking fun at the assault and saying that they would be happy to have it happen to them anytime.  For example, one woman said, "chris brown could beat me all he wants, he is flawless."

One of the biggest problems in our society is that we have a culture of violence where intimate partner violence is acceptable and abusers are not held responsible for their actions.     

What do you think we can do to stop the culture of violence?

In my opinion, Chris Brown is still the same angry and aggressive person that he was 3 years ago.  For example, instead of rising above and ignoring any negativity, he continues to lash out.  He took to his twitter after the ceremony to address his "haters".  He said, "HATE ALL YOU WANT BECUZ I GOT A GRAMMY Now! That's the ultimate F*** OFF!"  The tweet has since been deleted and he tried again with a slightly less offensive message.  But I don't buy it.  

For those of you interested in a more in-depth analysis of the original media coverage of the assault, I'll offer a publication.  This article was written by some fantastic colleagues at Boston University School of Public Health:

Rothman, EF, Nageswaran, A, Johnson RM, Adams, KM, Scrivens, J, Baughman, A. (in press).  U.S. tabloid magazine coverage of a celebrity dating abuse incident: Rihanna and Chris Brown.  Journal of Health Communication. Available online ahead of print.

If you or someone you know needs help, please call the National Domestic Violence Hotline at:  1-800-799-SAFE (7233)

Does pork consumption cause cirrhosis? Perhaps, if people become obese from eating pork

The idea that pork consumption may cause cirrhosis has been around for a while. A fairly widely cited 1985 study by Nanji and French () provides one of the strongest indictments of pork: “In countries with low alcohol consumption, no correlation was obtained between alcohol consumption and cirrhosis. However, a significant correlation was obtained between cirrhosis and pork.”

Recently Paul Jaminet wrote a blog post on the possible link between pork consumption and cirrhosis (). Paul should be commended for bringing this topic to the fore, as the implications are far-reaching and very serious. One of the key studies mentioned in Paul’s post is a 2009 article by Bridges (), from which the graphs below were taken.

The graphs above show a correlation between cirrhosis and alcohol consumption of 0.71, and a correlation between cirrhosis and pork consumption of 0.83. That is, the correlation between cirrhosis and pork consumption is the stronger of the two! Combining this with the Nanji and French study, we have evidence that: (a) in countries with low alcohol consumption we can find a significant correlation between cirrhosis and pork consumption; and (b) in countries where both alcohol and pork are consumed, pork consumption has the strongest correlation with cirrhosis.

Do we need anything else to ban pork from our diets? Yes, we do, as there is more to this story.

Clearly alcohol and pork consumption are correlated as well, as we can see from the graphs above. That is, countries where alcohol is consumed more heavily also tend to have higher levels of pork consumption. If alcohol and pork consumption are correlated, then a multivariate analysis of their effects should be conducted, as one of the hypothesized effects (of alcohol or pork) on cirrhosis may even disappear after controlling for the other effect.

I created a dataset, as best as I could, based on the graphs from the Bridges article. (I could not get the data online.) I then entered it into WarpPLS (). I wanted to run a moderating effect analysis, which is a form of nonlinear multivariate analysis. This is important, because the association between alcohol consumption and disease in general is well known to be nonlinear.

In fact, the relationship between alcohol consumption and disease is often used as a classic example of hormesis (), and its characteristic J-curve shape. Since correlation is a measure of linear association, the lower correlation between alcohol consumption and cirrhosis, when compared with pork consumption, may be just a “mirage of linearity”. In multivariate analyses, this mirage of linearity may lead to what are known as type I and II errors, at the same time ().

I should note that the Bridges study did something akin to a moderating effect analysis; through an analysis of the interaction between alcohol and pork consumption. However, in that analysis the values of the variables that were multiplied to create a “dummy” interaction variable were on their original scales, which can be a major source of bias. A more advisable way to conduct an interaction effect analysis is to first make the variables dimensionless, by standardizing them, and then creating a dummy interaction variable as a product of the two variables. That is what WarpPLS does for moderating effects’ estimation.

One more detour, leading to an important implication, and then we will get to the results. In a 1988 article, Jeanneret and colleagues show evidence of a strong and possibly causal association between alcohol consumption and protein-rich diets (). One possible implication of this is that in countries where pork is a dietary staple, like Denmark and Germany, alcohol consumption should be strongly and causally associated with pork consumption. (I guess Anthony Bordain would agree with this eh?)

Below are the results of a multivariate analysis on a model that incorporates the above implication, by including a link between alcohol and pork consumption. The model also explores the role of pork consumption as a moderator of the relationship between alcohol and cirrhosis, as well as the direct effect of pork consumption on cirrhosis. Finally, the total effects of alcohol and pork consumption on cirrhosis are also investigated; they are shown on the left.

The total effects are both statistically significant, with the total effect of alcohol consumption being 94 percent stronger than the total effect of pork consumption on cirrhosis. Looking at the model, alcohol consumption is strongly associated with pork consumption (which is consistent with Jeanneret and colleagues’s study). Alcohol consumption is also strongly associated with cirrhosis, through a direct effect; much more so than pork. Finally, pork consumption seems to strengthen the relationship between alcohol consumption and cirrhosis (the moderating effect).

As we can see the relationship between pork consumption and cirrhosis is still there, in moderating and direct effects, even though it seems to be a lot weaker than that between alcohol consumption and cirrhosis. Why does pork seem to influence cirrhosis at all in this dataset?

Well, there is another factor that is strongly associated with cirrhosis, and that is obesity (). In fact, obesity is associated with just about any major disease, including various types of cancer ().

And in countries where pork is a dietary staple, isn’t it reasonable to assume that pork consumption will play a role in obesity? Often folks who consume a lot of addictive industrial foods (e.g., bread, candy, regular sodas) also eat plenty of foods with saturated fat; and the latter end up showing up in disease statistics, misleadingly supporting the lipid hypothesis. The phenomenon involving pork and cirrhosis may well be similar.

But you may find the above results and argument not convincing enough. Maybe you want to see some evidence that pork is actually good for one’s health. The results above suggest that it may not be bad at all, if you buy into the obesity angle, but not that it can be good.

So I downloaded the most recent data from Nationmaster.com () on the following variables: pork consumption, alcohol consumption, and life expectancy. The list of countries was a bit larger than and different from that in the Bridges study; the following countries were included: Australia, Brazil, Canada, China, Denmark, France, Germany, Hong Kong, Hungary, Japan, Mexico, Poland, Russia, Singapore, Spain, Sweden, United Kingdom, and United States. Below are the results of a simple multivariate analysis with WarpPLS.

As with the Bridges dataset, there is a strong multivariate association between alcohol and pork consumption (0.43). The multivariate association between alcohol consumption and life expectancy is negative (-0.14). The multivariate association between pork consumption and life expectancy is positive (0.36). Neither association is statistically significant, although the association involving pork consumption gets close to significance with a P=0.11 (a confidence level of 89 percent; calculated through jackknifing, a nonparametric technique). The graphs show the plots for the associations and the best-fitting lines; the blue dashed arrows indicate the multivariate associations to which the graphs refer. So, in this second dataset from Nationmaster.com, the more pork is consumed in a country, the longer is the life expectancy in that country.

In other words, for each 1 standard deviation variation in pork consumption, there is a 0.36 standard deviation variation in life expectancy, after we control for alcohol consumption. The standard deviation for pork consumption is 36.281 lbs/person/year, or 45.087 g/person/day; for life expectancy, it is 4.677 years. Working the numbers a bit more, the results above suggest that each extra gram of pork consumed per person per day is associated with approximately 13 additional days of overall life expectancy in a country! This is calculated as: 4.677/45.087*0.36*365 = 13.630.

Does this prove that eating pork will make you live longer? No single study will “prove” something like that. Pork consumption is also likely a marker for wealth in a country; and wealth is strongly and positively associated with life expectancy at the country level. Moreover, when you aggregate dietary and disease incidence data by country, often the statistical effects are caused by those people in the dietary extremes (e.g., alcohol abuse, not moderate consumption). Finally, if people avoid death from certain diseases, they will die in higher quantities from other diseases, which may bias statistical results toward what may look like a higher incidence of those other diseases.

What the results summarized in this post do suggest is that pork consumption may not be a problem at all, unless you become obese from eating it. How do you get obese from eating pork? Eating it together with industrial foods that are addictive would probably help.

The NFL Super Bowl Commercial: Has Dedication to Player Safety Really “Evolved”?

In between a fantastic Super Bowl game, gallons of salsa, Madonna’s half-time show, and many disappointing sexist commercials, I patiently waited for the last third quarter commercial break. Almost a week before, the New York Times ran a story about a Super Bowl commercial which would address player safety. The NFL was taking one minute of its own commercial time (valued in the millions) to talk about its commitment to the safety of its players...a commitment which has been questioned over the years.

The one minute spot opens in Canton, OH in 1906 and follows one long kick return 100 yards (and 100+ years). The design of the commercial is well done as it follows famous players through the years. As the years progress (as documented on yardage lines), the viewers see and hear a discussion of key safety innovations. For example, we see no helmets turn into leather helmets turn into plastic helmets. We also see the addition of the facemask and the elimination of dangerous hits on players (e.g., horse collar tackles). The voiceover (telling viewers, “Here’s to making the next century safer and more exciting than ever. Forever forward. Forever football”) is provided by a veteran linebacker for the Baltimore Ravens- Ray Lewis.

In any occupational setting, there is concern that creating a culture of safety can be difficult, especially when veterans want things to stay “how they’ve always been”. Lewis has 16 NFL seasons under his belt and a reputation for playing with intensity. In many cases, he would be the hardest kind of player to get on board. Therefore, it is ideal for the Evolution campaign to have recruited someone like Lewis as a champion for its mission!

I also thought that the choice of words “safer and more exciting” were important. One concern about the increase in equipment and restrictions on hits is that it destroys the essence and entertainment of football. That essence being big men hitting each other, playing through pain, and ultimately being declared “stronger” and “winner”. So for Ray Lewis to promise that game excitement will actually increase (along with safety) is quite important for a possibly skeptical audience.

In addition to the commercial, an accompanying website was launched to provide detailed information about the history of the game and various rule changes. The website provides a very cool interactive timeline which outlines each decade. For example, the 1960’s bring “the world’s first Super Bowl”. It also brings fibershell and plastic pads, universally worn facemasks and the banning of grasping a runner’s facemask (1962).

From a public health perspective, I’m impressed with the quality and persuasiveness of both the commercial and website. A successful campaign was really necessary for the NFL, considering that it has been criticized in the past for its lack of commitment to player safety. Most notably, there has been much concern about the NFL’s protocols for dealing with player concussions (and the short and long-term consequences of those injuries). In recent years, we have seen a variety of public health strategies to address concussions.

They have developed and/or expanded safety committees to compile and analyze head injury data. We have seen numerous policy changes regarding both on the field play and the treatment of injuries. For example, in 2009 the NFL adopted a stricter statement on return-to-play for a player who sustains a concussion. The 2009 statement advises that a player who suffers a concussion should not return to play or practice on the same day if he shows any signs or symptoms of a concussion that are outlined in the return-to-play statement. In addition, the NFL has increased its use of financial penalties for helmet to helmet hits which are especially dangerous.

We have also seen educational materials for coaches and players regarding concussions. In fact, Pop Health critiqued the materials released in summer 2010 (“Concussion: A Must Read for NFL Players”).

While there is still criticism of the NFL’s commitment to player safety and there is always room for improvement, I think the “Evolution” campaign is effective. Reflecting over 100 years of football through the commercial and website, it is clear to the audience that they have come a long way. They have used various public health strategies to reduce injuries (i.e., equipment innovation, policy change, education campaigns, and probably the hardest strategy- culture change). In Ray Lewis, they have effectively retained a respected veteran to champion the cause. Finally, they have reassured the fans that the game will remain (and even increase) in excitement despite additional safety measures.

What do you think? Was the commercial just a PR opportunity for the NFL to defend themselves against recent criticism and lawsuits regarding player concussions? Or did you find it to be an effective reflection of successful safety innovation coupled with a sincere commitment to player safety moving forward?

The impressive nutrition value of whole dried small fish

When I visited Japan a few years ago I noticed a variety of dried small fish for sale in grocery stores and supermarkets. They came in what seemed to be vacuum-packed flat plastic bags, often dried. The packing was a bit like that of beef jerky in the USA. Since I could not read the labels, I could not tell if preservatives or things like sugar were added. Beef jerky often has sugar added to it; at least the popular brands.

I have since incorporated dried or almost dried small fish, eaten whole, into my diet. My family eats it, but they don’t seem to like it as much as I do. The easiest small fish to find for sale where I live are smelts. A previous post has a recipe (). I can easily eat 200 g of smelts, about twice as much as on the plate below; not quite dried, but almost so. The veggies are a mix of lettuce and cabbage.

As you can see from the macronutrient composition below (from Nutritiondata.com, for a 100 g portion), 200 g of smelts have about 112 g of protein, and 36 g of fat. No carbohydrates; or a very small amount of them.

Unless you misguidedly think that they will “give you cholesterol”, the macronutrient to calorie ratio of a plate with 200 g of dried (or almost dried) smelts is very good. Let us take a look at the fat content, below (from Nutritiondata.com as well), which is for 100 g of dried smelts.

The “net” omega-3 content of 200 g of dried smelts, after subtracting the omega-6 content, is approximately 4.4 g. The concept of “net” omega-3 content was discussed in a previous post ().

So, the net omega-3 content of 200 g of dried smelts is the equivalent to the net omega-3 content of about 20 fish oil softgels. (Yes, you read it right!) And you would get a lot more omega-6 from the softgels.

Not to mention the fact that isolated omega-3 and omega-6 fats tend to become oxidized much more easily than when they come in “nature’s package”.

Below is the mineral content (also from Nutritiondata.com) of a 100 g portion. Dried smelts are clearly a very good source of selenium. The significant amount of calcium comes mostly from the bones, as with many varieties of small fish that are eaten whole. Combined with the above, we could say that, overall, the nutrient content is high up there next to beef liver as a super food; a natural multivitamin, if you will.

Smelts, like many small non-predatory fish, are not a significant source of toxic metals. Many people avoid seafood because of concerns about toxic metal contamination, particularly mercury. The infamous incident that led to a major scare in that respect – in Minamata, Japan – did involve consumption of small marine animals. But it also involved years of direct and indirect exposure to very high levels of methylmercury from untreated industrial waste.

Other cases have been reported among populations consuming large amounts of whale, shark, dogfish and other relatively large marine animals with tissues compromised via biomagnification. Generally speaking, large predatory fish and predatory aquatic mammals are best avoided as food. If they are consumed, they should be consumed very sporadically.

Many people would say that a plate like the one above, with smelts and veggies, is not very appetizing. But I can really devour it quickly and go for seconds. How come? I use a special spice that enhances the natural flavor or almost any combination of “natural” foods – foods that are not engineered by humans – making them taste delicious.

This special spice is “hunger”. This spice can be your best friend, or your worst enemy.

Why Everyone is so Angry at Paula Deen- A Guest Post for "The Public's Health"

Last week I had the pleasure of being invited to guest blog on "The Public's Health". The blog is a collaboration between Drexel University School of Public Health and The Philadelphia Inquirer. In multiple posts each week, the authors highlight contemporary, historical, and ethical matters that challenge public health professionals.

For my post, I was able to contribute a "Pop Health" story which examined Paula Deen's disclosure of her type 2 diabetes and the media and societal backlash which followed.

What do you think? Do celebrities have the right to talk about their health information whenever and however they feel comfortable? Or do they have a social responsibility to disclose as early as possible- and to discuss it in a way that is most likely to improve the public's health?